These features - short duration of symptoms (<2 h), stereotyped semiology, and high recurrence rate - are all typical of TEA as opposed to TGA. Alternatively, it is recognised that a subset of ‘pure TGA' patients, largely identifiable because of atypically brief or recurrent attacks, develop epilepsy and/or respond to antiepileptic medication. Transient global amnesia (TGA) is a neuropsychological syndrome that involves a sudden and temporary episode of memory loss that includes inability to. In other words, the tumour might lower the threshold for the clinical manifestation of vascular and/or epileptiform events. epileptic phenomena, stroke, focal ischemia) have been proposed with no consensus on the exact mechanism 1,6. Such localised neoplastic lesions might result in abnormal electrical activity within medial temporal lobe networks, hence producing TEA-like events, and also compromise medial temporal lobe blood supply, for example in association with exercise, and hence predispose to typical TGA-like events. Transient epileptic amnesia (TEA) is a rare but probably underdiagnosed neurological condition which manifests as relatively brief and generally recurring. In some cases, the finding of a tumour is unlikely to be any more than chance concurrence, based on tumour location distant from memory-eloquent structures, whereas other tumour locations are more obviously of possible pathophysiological relevance. What is the clinical significance of these reports of TGA and brain tumour? Some reports fail to meet the clinical criteria for TGA whereas others have features which might be more in keeping with TEA. Transient global amnesia (TGA) is a clinical syndrome, usually occurring in those over 50, with as yet unknown etiology.
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